Search results

More filters
Clear

311 resources91 duplicate listings grouped

What’s searchable?
YouTubeJan 5, 2026
Some search terms matched

Dr. Purser explains the diagnosis and treatment of MTHFR (methylenteterahydrofolate reductase enzyme deficiency disease). Learn the genetic component of this illness (such as C677T or A1298C) and how it affects the body's inability of methylation to convert carbohydrates into ATP (Adenosine Triphosphate). Discover how the complex enzyme pathways of the Krebs cycle and citric acid cycle convert carbohydrate molecules into ATP molecules in the mitochondria. Explore the relationship of MTHFR to vitamin and hormone deficiencies while understanding how vitamin supplements can replace key missing (u

YouTubeJan 6, 2026
Some search terms matched

Your body’s ability to detox depends on one critical pathway—turning homocysteine into glutathione. But for millions with MTHFR or other genetic SNPs, this process is broken, leaving them vulnerable to toxins, inflammation, and cytokine storms. That’s why VARS Glutathione is such a breakthrough: it’s reduced, functional, and proven to lower harmful cytokines like IL-8 and MCP-1, giving your body the detox power it needs. After 15 years of research and real patient results, it’s the only oral glutathione that actually works where it matters—inside your cells.

YouTubeJan 5, 2026
Some search terms matched

Do you know what causes chronic fatigue? Dr. Purser explains the top causes of Chronic Fatigue Syndrome (CFS) and shares what steps to take to feel normal again. Discover why CFS is a symptom of underlying conditions and what the treatment of underlying causes of CFS includes. Dr. Purser dispels the myth of medication deficiency related to CFS symptoms. Discover the relationship of nutritional deficiencies, hormone imbalance, and poorly understood genetic causes from MTHFR deficiency to fatigue. Learn what contributes to fatigue when the absorption of Vitamin B12, folate, pantothenate, and oth

YouTubeAug 10, 2026
Some search terms matched

Why do some people in the same family get destroyed by black mold… while others are completely fine? Why do two kids develop autism while their siblings are completely normal? Dr. Dan Purser explains the hidden genetic issue behind both: a homocysteine problem that disrupts the transsulfuration pathway — the pathway responsible for making ~90% of your glutathione. Without enough glutathione, your body simply cannot clear toxins. Mold, heavy metals, environmental chemicals… they stay stuck. This is why some people get chronically ill and others don’t, even when living in the exact same house. In this episode we cover: How a genetic disruption in the transsulfuration pathway causes low glutathione Why most “glutathione” products on Amazon do nothing The only two patented forms that actually work Real cases of black mold toxicity and autism improvement Why checking genetics is more important than just checking a homocysteine level This is root-cause medicine — not symptom chasing. Learn more: Website: https://danpursermd.com Two Glutathione: https://twoglutathione.com Substack: https://danpursermd.substack.com Office: (801) 796-7667 Email: info@danpursermd.com #Homocysteine #Glutathione #BlackMold #Autism #Detox #RootCauseMedicine #PurserWellness #Genetics

FacebookAug 6, 2026
Some search terms matched

Are your B vitamins making you feel worse instead of better? Methylated B vitamins can be incredibly helpful for energy, homocysteine metabolism, neurological health, and many essential processes. But the right nutrient can become the wron

About search coverage

Search looks for your words in titles, descriptions, available articles, learning guides, and video captions when available. The closest matches appear first. Duplicate posts are grouped, with links to other places you can read or watch them. Books, chapters, and reports have topic guides here; the original files and full book text are not available in this encyclopedia. Subscriber posts include a title and public summary; open Substack to read the full post.