Let’s explore “IL-23”

This report overview covers the p19 and p40 subunits, interleukin-23, and the report’s distinction between the IL23A gene and the IL23R receptor.

IL23R is the gene symbol used for Interleukin 23 Receptor. This page connects its terminology with the topics in the Purser Wellness teaching library.

This report overview covers NAFLD, NASH, MASLD, MASH, hepatitis, the IL-23/IL-17 axis, and the report’s discussion of liver inflammation.

This report overview covers the p40 subunit, IL-12, IL-23, and the Th1 and Th17 pathways discussed in the report.

This report overview covers interleukin-23 receptor, JAK2, TYK2, STAT3, and the immune-signaling pathways discussed in the source report.

This report overview covers IL-23, IL-17, TNF-alpha, HLA-B27, spinal inflammation, and the treatment approaches discussed in the report.

A guide to a chapter asking whether IL-23 inhibition could have implications for homocysteine-related inflammatory pathways.

PURSER WELLNESSHow to Properly Treat Kids: Root Cause Medicine for Autism, ADHD, and Developmental ChallengesA conversation with Dr. Purser

… true. Here's another good one. Will glutathione help lower IL23 and IL17? This person has a double variant in their IL23R. Yeah, 23R. I deal with about four or five of those cases a week. some really prominent people. You'd be surprised. U…

PURSER WELLNESSMTHFR Mutations, Elevated Homocysteine & Autoimmune Inflammation: The Hidden Root Cause Most Doctors MissFrom the reading room

— A Guide by the World’s Leading Homocysteine Expert For more than four decades as a practicing physician, I have watched thousands of patients cycle through conventional autoimmune treatments that address symptoms but never touch the fire underneath. Rheumatoid arthritis flares, lupus rashes, multiple sclerosis relapses, Hashimoto’s thyroiditis, fibromyalgia pain—these are not random. They share a common, often overlooked biochemical pathway: MTHFR mutations leading to elevated homocysteine that supercharges autoimmune inflammation. This is not fringe theory. It is the clinical reality I have