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What’s searchable?This is a clip from https://danpursermd.substack.com/p/the-glutathione-industry-has-an-absorption?utm_source=youtube_shorts See the full video: https://www.youtube.com/watch?v=ZfFOnDVApWY #shorts #substack
Read matching passageDid you know your body’s natural defense against aging and inflammation quietly declines as you get older? Most people don’t realize that glutathione—the “master antioxidant”—drops with age, genetics, and high homocysteine. The result? More
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High homocysteine signals a broken transsulfuration pathway, leaving you unable to detox or convert it into glutathione. This shows up in conditions like PCOS and fibromyalgia, where patients often struggle with inflammation, pain, and even infertility. Genetics play a big role—there are 17 SNPs that can lock homocysteine in place—but vitamin deficiencies can make it worse. The real breakthrough isn’t lowering homocysteine itself but calming the cytokines it triggers, like IL-8 and MCP-1. That’s why VARS Glutathione, the only patented oral form proven to work, is so powerful—it lowers those cy
Read matching passageMTHFR is one of the most studied genetic SNPs, with the two main variants being C677T and A1298C. These changes reduce the body’s ability to process folate and make energy, leaving people exhausted and deficient in key nutrients. Specialized folates, B12 forms, and glutathione can bypass the blockages, and products like MTHFR Daily were designed specifically to fill these nutrient gaps so patients can restore energy and thrive.
Read matching passageHigh homocysteine often shows up first as widespread inflammation—pain all over, poor circulation, or even early warning signs of cardiovascular problems like stroke and heart attack. The best way to know for sure is through testing: a blood test can measure homocysteine directly, while a CMA with Redox uncovers the vitamin deficiencies driving it. For genetic causes, the DRP Genetics panel screens 15–16 of the 17 known SNPs that can cause permanently high homocysteine. If your levels are fixed by genetics, reduced glutathione and targeted B vitamins (like MTHFR Daily) are key to balancing the
Read matching passageWhat I see again and again is an inability to detox caused by failures in the Transsulfuration Pathway. This is your pathway that converts homocysteine to glutathione. If you have MTHFR or 16 other Gene SNPs, you will have problems with this pathway.
Read matching passageWhile CT scans are sometimes necessary, the radiation exposure they create can cause oxidative damage in the body. That’s why antioxidants like glutathione are so important—when taken in a validated, reduced form, they help detox and protect against the harmful effects of scans, heavy metals, and chemicals like gadolinium. Dr. Purser’s VARS Glutathione and Nayan Patel’s topical Glutaryl formulas are the only patented, validated glutathiones proven to actually work at this level, helping patients recover quickly from oxidative stress.
Just because you have EBV antibodies doesn’t mean it’s active—it usually means you’ve built immunity. The real issue? Most fibromyalgia patients have low reduced glutathione, which viruses like EBV intentionally suppress. That’s why symptoms overlap: fatigue, pain, immune dysfunction. Fix the glutathione issue, and you can support recovery on a deeper level.
Read matching passageHigh homocysteine? It’s not the number—it’s the cytokines it triggers (like IL-8) that cause strokes, kidney failure & inflammation. We found 17 gene SNPs + 12 vitamin deficiencies that drive it—and VARS Glutathione helps shut it down.
Read matching passageHomocysteine is nasty — but here’s the truth no one tells you. It doesn’t kill you directly — it triggers white blood cells to release IL-8 and MCP-1, causing inflammation, kidney failure, heart attacks, and strokes. Most doctors don’t even check it anymore, thinking there’s no fix. But there is. Whether from 17 genetic SNPs or 12 vitamin deficiencies (like B6 or B12), you can address it by supporting your body’s ability to convert homocysteine into glutathione.
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