ESR1 encodes estrogen receptor alpha, which helps cells respond to estrogen. Receptor signaling is different from the amount of estrogen measured in blood.
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What’s searchable?Explore dimethylarginine dimethylaminohydrolase 1 (DDAH1), asymmetric dimethylarginine (ADMA), nitric oxide, blood vessels, and related blood-vessel pathways.
Hmmm. If you’re diabetic you know that’s a big additional risk factor for death in this uhhhh current environment. Finally someone tells us why — hyperglycemia (high blood sugar) causes massive oxidative stress which in turns depletes your
NOS3 encodes endothelial nitric oxide synthase, which helps blood vessels generate nitric oxide, a signal involved in vascular relaxation. This pathway is distinct from a blood pressure measurement or a diagnosis of vascular disease.
…ular health, and differences between approaches to clotting risk. Understand the connection PAI-1, encoded by SERPINE1, helps regulate the breakdown of clots by inhibiting tissue and urokinase plasminogen activators. The 4G/5G promoter var…
Understand why homocysteine became a health topic and why lowering a blood number is not the same as preventing disease.
Homocysteine rarely gets the attention of cholesterol, blood sugar, or blood pressure. It should. When homocysteine becomes elevated, it has been associated with cardiovascular disease, stroke, cognitive decline, blood clots, and other he
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Instagram listingExplore volatile hypertension, autonomic regulation, monocytes, vascular inflammation, and how they may connect.
…ation teaching. The connection is to immune-cell traffic in vessel walls and other inflamed tissues, not a conclusion that an inherited marker proves active atherosclerosis. Compare the clinical picture with cardiovascular risk factors and …
Explore rs2236225, rs1076991, folate-dependent one-carbon metabolism, nucleotide synthesis, homocysteine, and double homozygosity.
Dr. Purser’s subscriber report on PAI-1 4G/5G, homocysteine, and brain infarct risk. Read the report on Substack.
The Overlooked Link Between MTHFR Mutations, Hyperhomocysteinemia, PCOS, Fibromyalgia, Chronic Inflammation, and Renal Decline — and Why Precision Genetic Testing May Be the Missing Step
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