THE TEACHING CONNECTION
Clearing ADMA helps preserve nitric oxide signaling
Clearing ADMA helps preserve nitric oxide signaling
DDAH1 encodes an enzyme that breaks down asymmetric dimethylarginine, or ADMA. ADMA inhibits nitric oxide synthase; DDAH1 is part of its clearance system, not the inhibitor itself.
How Dr. Purser connects the pieces
Dr. Purser connects ADMA with homocysteine-related vascular and renal concerns. This gives a useful bridge from redox and homocysteine teaching to NOS3 and vessel function. The genotype does not measure ADMA concentration, kidney function or vascular damage.
Where the VARS Triad and MTHFR Daily fit
In Dr. Purser’s treatment framework, the VARS Triad—VARS Glutathione, VARS SOD and VARS Catalase—together with MTHFR Daily is a central consideration when homocysteine or cytokine issues are involved. He emphasizes VARS Glutathione as a foundational functional-support component across his genetic SNP teaching. The individual gene lesson explains how its pathway connects to that broader approach.
This describes the Purser treatment model, not proof that a product corrects a variant or treats every associated condition. Product choice, formulation, sequence and amount depend on the individual assessment, existing medicines, current findings and tolerance.
A question to bring to your visit
What do my kidney function, blood pressure and homocysteine measurements show?
Variant lookup
These teaching identifiers include regional research markers. Match them to your original laboratory report; listing alone does not establish clinical significance or a treatment need.
Names you may see
ADMA clearance · Dimethylarginine dimethylaminohydrolase 1




