Wild genetic facts I learned this week that could actually be deadly

That's pretty fancy. What was that all about? [laughter] You like that little intro? I don't know. Yeah. Well, you're live. We're live now. I know people think I'm all the um this functional medicine. I don't know what a functional medicine doctor is. Um I'm

definitely not that. I'm just an MD. I just see um treatment options and and suggest them and some are you don't require pharmaceuticals because there's just because the approach we use. Um so it's interesting. Um okay, sorry. Sorry

for that aside. Um tonight we're going to talk well today we're going to talk about um some really crazy cases I saw um the last few days. Um, I keep thinking I've mastered all this stuff, but I learn

more all the time. Um, it's endless. Well, genetics are endless. Uh, and it's going to be that way. I'll be studying this stuff for the rest of my life. Um, and it's really fun to um to address it and figure this out and give people

viable options for major medical problems they have. Let me fix this. There. Uh, so I need to quit saying up. And I just set up. So these are some crazy genetic things I learned this week that can kill you,

which is always fun because you may not know you have if you have a max DRP with this. I designed that test specifically forine PMT, but there's so much more in it that I found uh interesting things that that are

really nuts. Um so do you want me to just dig into this and get started? Yeah. Now, if you want to give any more intros or shout out to anyone, just yeah, we're ready to go. Had a 40 year old patient roughly and and that's all I'm going to say about

the patient, but he had significant cardiovascular disease, which was surprising. I've seen several cases like this um over the years obviously because of homoyine, but um he had interesting findings. He had a um a double

homozygous MTHFD1 which shouldn't be something you think would cause cardiovascular disease. Oh, but I'm wrong. It definitely causes bad plaque buildup. Uh significant even. I

don't care how healthy you've been, how healthy you think you are. The MTH of T1, if you're double homozygous, uh and we check both of those, uh it causes mass vascular inflammation. And it's really interesting how you get rid of it. Here's the report I prepared for

the patient. Um, and then I realized yesterday I had two more of these. So, and now I'm looking for everyone. So, Jackson, uh, this is in the, um, the outlive your toxic genetics copy folder. Uh, you guys need to go back through

that again and look at that. Andrew, and you may want to start incorporating this. Yeah. So um double MTHF1 because how you treat it is um is kind of unusual. Um it

severely it severely impacts reproductive health and embryogenesis in women. Um severe MTHFD1 enzyme impairment leads to megaloplastic anemia. If you got a megaloblastic anemia, uh this is probably what you

have causing it and there's a way around it. Yeah, believe it or not, um you have a 15fold higher likelihood of developing choline deficiency organ dysfunction. So, it's a lot like PEMT, which we deal

with all the time. I get four of those cases a day. And um and it causes hypatic steattosis which is nash and muscle breakdown and muscle breakdown. It's crazy. So um central nervous system

manifestations like ticks um Tourette's uh mood disorders uh devel neurodedevelopmental conditions due to reduced SAM um and availability of neurotransmitter synthesis. So, um, so I

saw a patient with Tourette's and they had this. I'm like, there may be a way around it even though she's a little older. Um, I bet we can get rid of it. I'm hoping. So, that's Tourette's with the MTH1. Um,

so you've got to Here's the treatment plan if anyone wants to take a screenshot of it. It's the triad and oddly enough to reduce inflammation bars triad which is our bars glutathion bar side bars catalyst and these

take a take a screenshot of this. Can you do that? We can post it afterwards. That might be the stop moving if they might be able to. So [laughter] real quick before we continue on with these. So these are some of the top genes that you've just

been experiencing recently that you see are having horrible symptoms for your patients. Am I getting that right? Well, usually I deal with codicine issues all day long, but there are definitely other things other than sodakine issues. Obviously, there's millions of genes out there and we

really specifically look for a a really hard set of problems that uh comprehensive cytoine panel I developed and it's done now. And so let me mention that while we're here, that satic panel is done, people. Um, we're going to make

it available. What do you think? October 1st. Yeah, probably by by the end of the month. So yeah, let's plan for October 1st. No, no more messing around. No more, you know, no more corrections. Not going to add anything else to it. Has all 90 of the cytoines in it. Uh, MaxGen's been

really good. This is such a comprehensive test. um and that we had to validate it and everything and go through it, run through it. Um so we know our material. Uh so I can't believe some immunologists or some immunology

group hasn't developed this test. They should use it um because it answers a ton of questions. Not all cytoines have a biological form either. And if you have multiple cytoines, two or three or four cytoines going off, what do you do?

Um, so there are seven big studies none that we've ever done that show that that a functional glutathione and there's only two in the universe. Uh, the glutarel that Nan Patel and Chinron developed in La Habra and I helped helped a little with that. Um, and

they're my friends. Uh, we were on that that pituitary endocrinology cardiology team at USC together and good guys. They own Central Drug and La Habra. Some of you may already know them. Um, so or have been in there if you're in SoCal.

Uh, but um, literal will suppress cytoines. So will var glutathione. So they're it only two patented validated functional absorbable reduced stable glutathions in the world.

That's where we came up with that law that law of antioxidants working with those guys. Um, so, um, but so that's an option. We use it a lot as soon as there's not a biological answer for it. And they'll try you on everything. I saw a lady on on $1.3 million a year in in u

in sodaki and sorry, biologicals. Uh, but they weren't for anything she had that I could see. I'd figured out what she had by happen stance. They were just guessing. Uh, unbelievable on a guess

really. I mean, what is and she was they hadn't helped her. They were improving her life. So, um, so yeah, I see that stuff all the time where someone's on 300,000 a year in some cytoine, but it doesn't really do anything for them. It's not

appropriate for what they have. Glutathions just suppresses the cytoine production. And the biologicals are incredibly helpful and a gamecher for a lot of patients obviously because they can block the absorption of of these cytoine to the receptor site or vice

versa. There are actually floating around receptors 23R is one of them where they just float around and look look to find a 23 to attach to and they go off like crazy. Uh that's a that's a bad deal. real quick question for you and I don't want to get you too off

track this um but with with these this like cytoine it's kind of like a cytoine storm that a lot of people are dealing with. Yeah, we all heard about it during Yeah. Yeah. During that time is it almost like an autoimmune disorder that's happening?

An autoimmune disorder. It is the strictest definition. Patients tell me they have autoimmune disorder. I'm like which one do you have? They don't know. It just causes inflammation. Like it's a cytoine problem. Mhm. these little proteins God gave us to go and um then you squirt out little tiny

microscopic amounts from your white blood cells to go off and chew up old scineesscent cells that need to be removed and keep our body clean and all this and that. But it and so they go out and do their job. But if they're homozygous like turned on to the max 247, they go out

and remove you. They remove everything that in its way and so if like IL8 which is what homoyine causes goes out and cleans out your kidneys but IL8 the one that homocyine causes or if you're homozygous for some reason seen that a

couple of times it is ugly it removes your kidneys shuts that a number one cause of kidney failure um so that's why you'd want to get a a cytoine genetic panel to see if you even have any of these genes or any homozygos does not psychos.

Yeah, that's why we developed that panel because we only I in this my max DRP we put the top four sodicons out 23r 6 tf alpha and I can't remember the other one but and we look for all the homoyistent

causes. So um and um you want to find out what those are. So I diagnose these all sixes all 23 hours all the time. Um I mean I have prominent physicians I take care of that um that have been to

all the specialists they're they're with universities whatever and they have major uh inflammatory conditions a lot of people do why can't doctors and yet they can't get help for it just like you and it's a um it's a cytoine problem like it's 23R or is 6 or something like

that is 6 is more known for causing bad cardiovascular disease 23R it causes joint destruction, all this kind of stuff. And they're worse than those. So, we're going to talk about those really quick. So, this person had um M MTHFD1

double homozygous, and you need to treat it with with B2, with TMG, with glutathione, with not because there's cytoine involved, but it removes that vascular inflammatory component, and there's nothing else that do it. A

statin won't do it. a steroid won't do it. Um, and they also probably should be on a baby aspirin today. That's up to them. A lot of patients won't take it and it's kind of I don't it's kind of redundant if you're using glutathione for that situation. So, and there's

others. So, um, cool use of TMG right there. If you have this in your MaxG or any other test, email us and we'll send this to you. If you watch this Facebook live, yeah, we'll send this, we'll send you

any of these you want. So these reports, you can kind of look at them because they can save your life. Um, the next one I'm going to talk about is hemocchromattosis where you have an iron absorption problem. Let me tell you, that's not just a a yucky condition that will kill you

because it causes that if your iron ever gets high in your blood, it causes starts causing mascular sorry massive oxidation in your cardiovascular system. And so, um, here's a HF report that I I created for,

um, homozygous HF HF gene H630D mutation. actually um and um it's charact characterized by prolonged symptomatic asymptomatic pre-clinical stage where you have a lot of uh high

serum uh iron indices. So if your if you come back if your iron panel keeps coming back with high iron, you've got it you got to lower that. It's absolutely

it's just absolute that you got to lower it. Um because it will cause vascular inflammation. It will kill your kidneys. It cause heart attack, strokes. Um and the symptoms are vague. Chronic fatigue, lethargy, arthralgia. Um and if you get pain in your second, third metacarpo

felonial joints right in here. um my hands. This is from playing volleyball and stuff in college, but um uh that's a a sign of it's called iron fist. Um and you u man it'll destroy your liver and

your um and your heart. So, and you throw it in with the homoyine problem. Good lord, man. So, you got to keep an eye on it. So what we're advising people to do now with it um is um

is um check your CBC weekly even month well okay monthly at least you can do it through direct labs during the lab test now walk-in labs cheap also get an iron panel at least probably once a quarter if not once a month keep an eye on that if you don't it's going to bury you uh

and you'll have horrible inflam all this um and um and um you usually get phbotomized which is also called venuction but you have to get phbotomized only if your levels are high. There's nothing

else you can do for it. It's not going to magically go down. Um though um um queretin um can help it. Bars triad can help it a lot. You want to keep the inflammation down. It's critical. Not

trying to sell your product. I'm trying to keep you alive. Um and so um again, if you have hemocchromattosis, get this report. It's not it's not going to tell you. It gives you options for things you can do naturally because I don't know how you

do it un there's no way to do it unnaturally. Get full bottom and and um and so also you should get iron panels C in CBC on a regular basis. Can you get those through? Pay for them with a credit card if you have to. If your doctor won't do it, they should order it

to go through your your insurance. Um, and a iron panel. Uh, you should get a standing order for that at least every every two weeks or every week if you have to get it so you can get it down. Get and a standing order for

photmization and a standing order for a CBC. And if they won't do it, you got to do it. you're responsible for your your own self a lot of times because doctors don't know how what to do with this. Um this report is highly validated and um I

give it to a lot of patients. I'm going to be a lot more aggressive about um about it in the future too. Um does that play well? You guys they seem to understand that and don't just say you have it. You can get genetically tested to make sure you

have it. Our test looks at that. So um um and that cytoine panel looks at 90 different cytoines. That is not a cytoine panel. That's an iron inflammation disaster waiting to happen. Um

the next one is um is is really odd. this person that um had a a combined MAOA, three of them, three of them and MAOB

right here, homoasty report. Well, that's again another freaking cardiovascular nightmare. Um the primary cardiovascular consequences uh is severe adinuric vascular hyper reactivity. So your blood pressure is going up down doing all

kinds of weird things and you you feel all this inflammation. You get headaches, severe throbbing headaches, suddenly palpitations dangerous to acute hypertensive crisis. Um, in addition to hypertension, excess circulating serotonin and catakolamines accelerate

arthro athrombosis and microvascular eskeeia. PL store vast amounts of serotonin in a hypopunctional mao state. elevated serum u or plasma serotonin binds the platelet receptor promoting hyperagregation and

clots. Lovely. So um so you need need to be all over it. And it goes through the the drugs that can be prescribed for that now if you have it. Also goes through um natural fibbrronolytic therapy. You need nattokinness 10,000

fus a day. Um and I don't like lumbarinist but get the nattokinness. I get it off Amazon. The omega-3 fatty acids. Um my favorite is uh is the you can email us for all this design for health. Um u uh omega val synergy is the

best one out there to those and you also need curcumin uh because it inhibits the cox one cox 2 pathways and down reggulates that that cell adhesion problem. Uh you also need ribboflavin B2 um to uh to um you do high dose

riboplavin supplementation 100 to 400 milligrams a day um and that helps you um prevent it from um from clotting. You also need quin which stabilizes mass cells and the only validated quin I know you guys probably all tried quin for

high histamine problems but queretin there's only one validated one out there and it's um it's designed for health reserverrol supreme yeah it's in there it's the corsetin in there is the only validated um a corsetin in the world that I know of I

gave that talk at that amm um you were came and recorded ed me at that meeting in was that like March or April of 23? Uh 22 I think. Yeah. And I looked everywhere for validated coretin. Um because it was

critical for for dementia prevention. Um oh yeah all these things probably definitely increase your risk for dementia too by the way. Um so um you also want to take lowd dose aspirin. One baby aspirin. I like the

the Bayer um low dose baby aspirin 81 milligrams. They suggest beta blockers stuff like that. Um I like the nattokinise like the omega-3s. I like the curcumin. I like the B2. I like the bars triad. If you

have that condition or tri is not the answer for everything, but it's the answer for a lot. If you have a a glutathione, if you have a high enough glutathione level, I've read in a couple different papers, they don't think you'll ever have a heart attack or stroke. Um, I don't know if that's

accurate, but it's sounds pretty accurate from what I know. So, anyway, interesting stuff. Um, so yeah, that's a coma and two MAO on that maximum we developed. Uh, email us and go look it

up. email us and we'll send you this report because we're learning new stuff every day. Um I wish I knew it all. I wish it was born knowing it all. I'm not an AI even they have to learn stuff. So um so the other the next one is PI1 4G 5G see

it all the time but that with himer that with hyperhomocyinemmia which I see 15 patients every genetic patient I day has a homoyine problem. So look at my textbook on homoyine that just came out. Also there's a new book coming out where I really

consolidated I shrunk each chapter down. It's like a handbook for homos for homoyine that contains all the key points. Is that ready yet for publication Jackson? I don't think so but it's getting close that's in it. So you guys better check

it. It's Yeah, we just need the cover. The cover. Yep. Yeah. But if you throw in PA1 4G 5G with homoyine, you got a major stroke multiplier. PA1 PA AI1 4G 5G causes stroke many strokes

and you throw it's a clotting disorder and you throw that in with a homoyine um you're in deep trouble. Um and um so and again get the report lowd dose aspirin methylation support. Um

they suggest a pexaban or rivo rivroaxaban. So I'm going to throw those in there too. Um but they cost around 400 to 600 a month and carry severe risk of major side effects. Recombinant tpa.

uh they I find most strokes uh are not being treated post uh stroke correctly to prevent further stroke because they've not done any genetic testing that really matters on and if they say we don't know what caused it we're going

to give you this you know what you better damn well know what caused it. So um back to the bars triad the nattokynese um and those are really good options. So anyway, the report's pretty detailed. So

if you have PAI1, 4G, 5G, and a lot of you patients do, and I told you then that the triad, no, glutathione would offset it. It's still true, but um you may want to, and I also see a lot of you get mad when I suggest a baby aspirin

because aspirin is somehow evil. Remember, aspirin's natural really. It's been around for a million years. It's a It's from a tree bark originally. Um, but yeah, for that you better take a baby aspirin every day. Won't hurt you

and hopefully prevent you from having a stroke. Am I boring everyone? I'm probably boring everyone. No, I find it fascinating. What do they have one person watching? [laughter] No, they they're enjoying it, too. They keep asking where to where to

get these reports. And so email info@danpursermd.com and we'll start getting them to you. Um but yeah, send us your your genetic results to or if you know if there's one specifically that you're that we're looking at, let us know that you have it and we'll make sure to send that on to you. Yeah. Um so the next one is the one

of the two most common uh cytoines I see every week. I take care of about four or five P. Yesterday I had three. I had three yesterday with IL6 interlucan 6 is a is horrible. Uh it's one of the most common cytoines. It causes really causes

really bad cardiovascular disease. That's what that prop for discussing this. Um if you know you have a homozygous you match bear IL6 as for this report. I've got a whole lot of reports and diagrams and all kinds of stuff on it. Um it also causes nash and

cerosis along with PMT that's just deadly. That's but again glutathione we tend to use the triad because you're going to have so much pain inflammation in your joints and everywhere else with this condition um that uh glutathione

suppresses all cytoines but act is the drug you want to get on too if you but I can't I'm not going to prescribe it. I'm not an immunologist. These are all autoimmune disorders. Um so uh but but I give all that information in the report

so you can go and ask for it. Um good luck on that. Um but um yeah, and hopefully your insurance will cover it. Uh but it cover does it causes migraines. I keep getting these patients with migraines. They're all they all say

they have fibromyalgia. They're told they have fibromyalgia. I let me tell you I know more than anything about any human on this more than any human on this planet about fibromyalgia. We have that 10,000 plus patient Facebook page end fibromyalgia with natural options.

Um these people have cytoine problems. They all have a homocyine problem. They don't know that they do but all the ones who tested from there do and so that alone can cause cytoine problems. But I would suggest all those

people get that good lord that'd kill us, wouldn't it? Get that um get that report on um get that 90 cytoine panel and check it because it contains every cytoine. Um it's going to be the most thorough

cytoine valve ever. And then you know if there's a biological for it, you know what your what your weird problem for your fibromyalgia is caused by. You'll know why you ache and hurt. you know, why your dad died from a heart attack at 33, all that kind of stuff. Um, or why

your joints are being destroyed. Um, so it's not just arthritis. It can be cytoine related. You can't you can actually recover some of that functionality if you approach it correctly. Trying to stay natural. Um, with the with this fibromyalgia

connection, does it play with the copper? Does the copper interact with the cytoine? Yeah, I still think I'm right about copper being a heavy metal problem. They can't clear it because they have a homocyine problem and if they have that net not enough glutathione that nat net two which we looked at

because it causes fibromyalgia. I have a patent on that copper balance because it makes that two work. I mean it it does what two should do. So, um, yeah, if it wasn't gone, homozygous and

out of there. Um, so yeah, I think I'm still right about that. But some kind of heavy metal problem, but it fits with a homoyine problem. Homocyine problems explain why like if some members of a family have it, some don't. It's why

some people suffer badly when they move into a mold infested house. And the other the like the dad and the the two other girls are like we don't have any problem while mom and one of the sons and and and the other daughter are just dying from something and they coughing,

sneezing, feeling horrible, super sick. It's black mold and that's why you hear all these people going they have black mold problem. No, what you really have is a homoyine problem. And what that means is you have a toxicity problem because you don't make enough glutathione to clear black mold. So you

have to borrow it. You have to take it and people keep taking row just we're talking about medical issues really or genetic issues here. Row is not for that. It's row I'm I don't think they have any validation or patents. Haven't

seen them file for any patents. So they have great marketing. They have great marketing. That's really good. So yeah. Um so but uh you'd want to take a glutathione that works to save your butt. So uh either the VARS or the glutarel by nine in them. You can get

that at our office too just because they helped with all the work on it and did the and did the u a lot of the uh pre pre-clinical trial work on it. Um you can track is six you can see it come down super you can track eight if you're

all eight from homocyine. You got to watch for flares with those conditions. I spent a lot of time teaching each patient about watch for flares. An infection in grown toenail, a earachche, a sore throat, a virus can put you into a flare for a couple weeks while it's

ongoing. On and on we go. The most common cause I see for bad flares are teeth infections and they they're disastrous because they not only cause your tooth problems but they cause a flare and you can die from

a heart attack, stroke or kidney failure all of a sudden. Hypertensive crisis. So um I tell everyone to get a blood pressure machine, check their blood pressure. If it's going up you're in a c you're you're in a hypertensive crisis, especially if it goes up fast. and that's caused by a flare. But like and

so eight could be really high. Um we track those and a lot of my patients track those. I tell them to uh if they have I6 I tell them to track that because it's more likely to go up than IL8 and it's easier metric. Um I 23R you

can't check a level on. No, that's what we're going to talk about next. 23R causes all these horrible gut problems, inflammatory problems in your joints. unknown. M we go if you know you have a 23r get this report. Um

it talks about the there's some protective variants and but there's a hyperinflammatory hyperfunctional inflammatory variant and there's diagrams [snorts] in the report. We talk about everything on it. It's a long report. Lots of diagrams, pictures,

everything's referenced in these reports. Um like there's nine references in that report. Um, and it is on that new panel, too. But I I I don't know if you want to get the MAC I'd probably start with the max DRP first, but if

you're just curious which side of county is abusing you, um, call and and pre-order that report or get on a list so we can email you um, and when the report's available. So, um, so the next I saw yesterday was

a I6 2023r combination. Hellish. It's horrible. These people are so sick. So, um, I got this diagram for it. Uh, I've got a paper for it somewhere. I just didn't have time to

pull it out. But what those two things do together are just nasty. Um, so yeah, people I mean I get I see people all the time with two or three cytoines. Um, and so that have been diagnosed. Uh,

I don't know how they find them out. I get sometimes have to do a a 14 cytoine blood level test. Um, that's the only way I've been able to find out other cytoines. Uh, so I don't know how their doctors are finding out. um that but now

our new cytoine panel will find all of them out. So um I'm not trying to say something I'm trying to help you. So and I've worked three years on that on that sine test. So um we need to create a book Jackson of just all those cytoine reports.

Yeah, that'd be great. kind of like this a book of these reports and just have all the the great information around it, what symptoms it causes and what good treatment options to deal with it. We can do including your biologicals and

even the glutathione sod catalyst are really good adgivant treatment with these with the u with the biological. They don't interact. They they help each other. Um, so you get really good care and if you get in a really big flare,

they don't go up on your biological, but you can go up on the sodicon. I mean on the uh the the triad. We go we always say 1284. That's what I say. 12 bars glutathione a day, eight sodalis. It's 529 a month. If you want to know the

number on that, but most of the time you only take it a week or two. You should be able to figure things out in that time and and bring down that flare. You're like being a diabetic who can't get infected because they get in deep trouble. That's what having these cytoine problems are like. So every time

you get an infection go, "Oh my god, what do I hurt all over? What is the matter with me?" Yeah, it's a cytoine issue. And you get in a flare of stress, trauma, or an infection. Mo most infections being the most common. then

you'll you'll um the sun will kind of blow blow through the roof and and that's when you're at highest arrest for dying or or dam high damage. So u that but we found that 1284

always suppresses it. I don't care how bad the flare is. At least until you can figure this down. Look at my reviews. Yeah, I think too it's good. A lot of times people get concerned or worried

that they have to stay on a high high dose of glutathione sod and catalase for a long time. They're not in a flare. They come down to like they figured out they're like okay I have no pain if I take 642 six glutathione four sodas or it varies

421 642 8 83 you know 8 86 three whatever. So I I want to make two quick little notes about that. Our VARs catalace product, the ingredient in there uh called

alevity is that Nerf 2 activator, but they just finished a couple of human clinical trials on that ingredient showing the increase of indogenous glutathione in the body up to 40%. Um which was pretty cool. And then also they show

of course we don't know the genetic status of the people in the test. Yeah. Um, but it also showed that it was lowering a couple different cytoines. And I don't have that data in front of me, but I think it was IL6 and IL8 where in their test they were showing showing the cytoines going

lower those all the time. Yep. Yeah. It's it's the science. It's not magic. It's the genetics. Genetics say glutathione should do this and it does do this. But everyone with homoysteine problem, you've lost 90% of your

production before you're born. That's the definition of a homoyine problem. You're not converting homoyine to glutathione. You get 90% of your glutathione in that transuleration pathway. So, we got it right there.

There it is. Wow, that's fancy. Yeah, we I did that. Yeah. Now, I always I always love bringing this one up just because it's the perfect visual for that. Um showing exactly the process that has to go through even to just make glutathione down in that bottom right. But all these

other genes, all these nutrients have to be there and functioning for this all to work. To be actually I think it's more complicated than this, but still it's it's the simplified one. Yes. I know it's more complicated.

Yeah. Reach out to info danpermd.com. Uh if you have if you know you have a a cyto problem uh and you want a report, I've got a report on all of them. Um, so we'll send it to you. Um, and if you

need more than that, you want to you want to do further testing or you want to find out if you have a cytoine problem, reach out to us and then I'll guide you. Um, you can go find an immunologist too at university or whatever near you and get help that way. But we're helping you our way. Um, and

uh, figure out what you want to do to get rid of your pain, inflammation. The good news is none of this is permanent. the next four to seven years I think at most with the new studies at Harvard and all that um we're going to be able to modify your genetics and get rid of this

but knowing what genetics you want to get modified would be really helpful so instead of guessing um yeah so um you just go in and get an IV for an hour hour or two um lay there relax a book and then you go out in the

next few days the uh your gene things get modified to back to normal. You're going to happy healthy life. At that point, you wanted to take this stuff. Um, the day is coming.

So, uh, we had a couple questions if you want to do them too real quick. Go ahead. Someone was asking Oh, I can show them to you. EDS and cytoine storm. Is that Eller's Danlos? Yeah, they're Yeah, Eller's tendless is highly over inflated. It just means lax

tendons. It doesn't cause it doesn't cause any cytoine anything. You just have lack you're born with lax tendons. Got to be careful the weight you lift. There's a lot of false information about EDS. I see it all the time. It's not not really

much of a thing. Some people don't even know they have it. Most people don't know they have it. And then they go look stuff up and go good god is g be happen like no it's not no doesn't do that no cytoine storms for mess you have some

other reason you're having the cytoine storm probably homoyine um and uh the homocyine is kind of mandatory for you to have a cytoine storm because if you can't convert a homoyine to glutathione you have no way

to suppress that cytoine storm naturally if you do not have a homoyine problem and there's 22 snips that cause it. U grab that handbook when it comes out or grab my textbook if you want to know. There's a whole chapter on it. They used to think there are four or five. They're

22. If any one of those 22, you're in deep yogurt. You can't make enough glutathione to get you out of trouble with the cytoine. So, right there, you're going to have massive inflammatory problems.

Um, you kind of mentioned this before, but do you think they that someone should get the sodicon panel even if they've done other genetic testing? Sure. They haven't done this testing. I don't know that anyone's ever made this available before. No, even developing it with MaxGen, they

were surprised and they were working on their own cytoine panels already, but uh yeah, they said that no one has done this before. Yeah, my knowledge base is huge. I mean, it has to be for what I do every day. I deal with that crazy inflammation that no one can figure out. Pain,

inflammation, destruction, P. Yeah. Or DIP, DIP, because you feel like a dip because no one can figure it out. Um, do you know anything about burning

mouth syndrome that's not nerve related? Have you heard of that? Yeah, it's usually B2 deficiency. Okay. a CMA with a redux. It'll tell you if that is indeed it. Yeah, because I had that Olympic u athlete who's

two-time all-American, all that, who had severe B2 deficiency, mouth was burning, hands and feet were numb. Um got sent home from the Olympic training center because kept treating him for a hand foot and mouth disease. You bunch of knuckleheads, do a vitamin panel on him. Good god, it was a horrible B2

deficiency. took me nine months to to get him normal where he could use his hands and feet. His grandmother was taking care of him and I'm not going to mention his name or what state he's from or anything like that.

Um would all of these cytoine and homocyine related genes would this be related to juvenile idiopathic arthritis? Is absolutely the cause of it. These cytoines are Yeah. Did you hear me? It's absolutely the cause of it. And they they're fussing

around with it. And you can actually with this test you it's just a swab. You can figure out what those genes are. There may or may not be a biological for it, but you can stop the joint destruction at least. You can figure that out. They'll develop them. And you can stop the joint destruction for the

most part with glutathione. Even if we're a kid, we do this all the time. Yeah. Yeah. I was I was at the dentist this morning talking with my dental hygienist and explaining a couple of these different genes and things to her

and she was so fascinated by the idea that like just take glutathione for example. Everyone makes glutathione to some level one way or another. But when we see Exactly. And so we see these genes and we just see much less efficient makers

of glutathione of all these different all these different nutrients, different things we use. It's all this balance of efficiencies. And so some people are winners in in life. It just they get have perfect genes and have perfect nutrition, whatever. It just it works

out great for them. Many many other people are not that way and just are always behind their entire life until they start supplementing, until they start getting on the right forms of these things and helping their genes, helping their body do what it was always supposed to be doing. Yeah. I love that these and I'm going to

bring up an old complaint. I've got these doctors who you say you have you go in and say I have an MTHR problem doctor. It's going to cause home assistant problem or whatever. They'll go that's not important. None of that's important. That's not a thing. That guy's scamming you or something. Ah,

read a book, dude. Look at Go to PubMed, look something up, you dork. So, just really I mean just it's insulting. So, I've [clears throat] got a textbook out on homoyine. Read the textbook. Read the references. Read the validation. Then

come at me. Bring it on. Yeah. Um, okay. We got a couple more questions. They're gonna say it was only trying to sell you something. Yeah. I happen to have the one of the only functional gluathons. So, MCAST is someone asked

for talk about MCCAST, please. Um, MCCAST is caused by a number of different cytoines causes the histamine or it could be a DAOA histamine type response like that

um like that MAOA condition that guy had. So um and he did have a lot of MCAST symptoms. And someone on Instagram was asking about liposomaal glutathione. Not to

bring it back to glutathione again, but just your thoughts on that. I don't know what that means. That's I mean there's there's 70 on Amazon that don't work. I mean they're not validated. If you're if and I'm I'll stand by that. Really prove you have validation. Do you have a patent? If you

have a patent and you probably valid and probably violated someone else's patent that's already been done. There only two out there that have been done. So really I mean there's a lot of old patents that didn't work that people got. We know through validation everything else that what works what doesn't. Quit using

garbage. I mean you buy some glutathione. It's not that's not the trick. The trick is the the um reduction the redux status. So and that's what we have the patents on. Um we keep our

glutathione uh stable de deoxxygenated because oxygen will will um will ruin glutathione. So we doesn't see air and the water we put it in um eventually it has to have no air in it. We remove the

air the free air from it. On and on we go. They haven't gone through that and we got a patent on all that process. So good luck. Yeah. Yeah. Just because something says liposomaal. Mhm. If you have one that works in it to us, we'll assume. Okay.

Yeah. I liposomaal should be the one of the optimal delivery mechanisms, but it all there's so many factors in the manufacturing process to make it even just a reduced product or if there if it's even a functional liposomaal product. So, it's a good question, but

definitely stick with vars glutathione or glutil. We have a lot of that and patent work. So yeah, you don't see it. It's invisible, but it's there.

Uh any experience with PBA, primary progressive aphasia? Yes. Um it's usually some toxicity issue that hasn't been diagnosed correctly because they have a homoyine problem. Uh try them on glutathione. So you can shut it down and and it often

reverses from what I've seen. Good question. Shoggrren's disease is usually not. It's usually low testosterone in a woman. You're postmenopausal.

You quit making testosterone. Um and so you're dry down there, you're dry in your eyes, you're dry in your mouth. It's just lack of testosterone. Testosterone women also has five big cardiovascular benefits. But but the stuff you feel is dryness if your

testosterone is low. That's my opinion about sugars. There are a lot of cytoine related disorders. Thank you uh Lynn for saying that. Yeah, it will change lives.

It's really good. Yeah, you can take it regardless. Probably you need it, but this is I don't want to say that blanketly, but yeah, I take two or three at least two a

day. Someone ask them regardless of genetics. Yeah, two a day. Majority of people, especially as you're aging, most likely it's going to be beneficial. Probably the most anti-aging. Uh, I read this by I think it's the NIH or somewhere that's the most anti-aging

product um if for you know for one that works which they didn't know that there was one that works but out there in the marketplace. So most anti-aging thing you can do is help your glutathione level.

Um I don't know if you can read this whole one if it shows up for you but um someone using the glutaril for their child that worked really well. Yeah. Yeah. We've done that several hundred times now. U because kids with I'm gonna be really careful what I say

because I'll get shut down. But kids with um um I don't even know how to word it. That a problem. Detox problems. Detox problems. They're getting worse and worse. They used to could talk. Now they're five and they can't talk.

They're they're just get really frustrated about everything. Yeah. That's because their toxins are building up because they have a homocyine problem. Um, and again the one thing they can't make which is glutathione. We usually use glutal but um in that situation we have a liquid glutathione

that we're going to come back and make again here shortly but um um it's it's true it works. Doesn't always work because there's other damages that can occur. Um but uh it mostly works.

Yeah, glutaril especially for young kids is great uh because you can do such a small dose just a tiny spray on the bottom of their foot and just to give them a nice little start with their glue. I would do a drop on the top and rub it in with your hand. Go really slow with a Herxheimer. Even

little kids will. Um the other problem you've got um let me think here. I was gonna say something else. Uh, no, no, I can't think of what's called um, yeah, but yeah, but they have to take it

forever because they can't make it. But forever is four to seven years now. Um, then change their genetics so they can make it. Yeah. Um, someone was still asking about MCCAST. There's a really, really good

video from Dr. Perer on YouTube um, about MCCAST. Just look up MCCAST and SAD S O that'll give you a little bit more information about the information that's in our SAD capsule and our rejude.

So yeah, but that's a triad because work together. Their husband and wife. So yeah, you need all three. Call is the angry mother-in-law telling them what to do.

That's how it works. [laughter] some. Oh, that was a really good question. Dad, did you like that? But chief genetic guru for my neighbor. Uhuh. I noticed that. That's pretty good. [laughter] Okay. Well, I think we should wrap it up for now. Do you have any other shout outs or anything else you want to give to everyone? Email. Don't be dorks.

Info@danpermd.com. Email us if you have questions or thoughts or you're like, I want this. I want a report. Do you have a report on this or that? And I we have I've created hundreds and hundreds of reports. So they all have research behind them. Um

the office is already getting emails and they're already emailing me asking about where what these reports are. So we got to go help help them send out the right ones. But storm lovely here for you. I I I keep in mind I know

I love everyone that goes takes their genetic information to an AI. Oh, that's smart. I'm about I'm at least probably 8 to 12 years ahead of that AI and most doctor and they're being sure they're being given the information not by doctors but by just dummies I don't know

anyway quick a quick comment on that real quick um snipedia snipedia um there's several different databases out there that literally everyone uses when dealing with the genetic snips um and So,

everyone pulls from the exact same data pool and there's so much contradictory information out there. One person does a study on a gene that says this and then another one says the exact opposite on that same gene. Extremely contradictory out there. And so, you can ask any AI and it's going to get very complicated

real fast. Um, so definitely I mean if you have genetic questions, come ask us. Yeah, because I I'm in the trenches. I see these patients every day. Um, I have this wide open genetic practice. I never seen him with like three arms or three eyeballs or

I never never see anything really cool like that. I see if you're out there could you could you connect with us but um but uh just I want to take a picture but um but yeah I see people with inflammation I see 15 cases out of 15

patients with homoyine problems every day. So um uh in four I'll have PMT and then so it's all right in my face and now I realize I'm finally people are listening

and coming to me with really bad inflammation and then I find cytoines but all you people that I'm like you need this test because you don't have those top four you got others now's your chance we'll find it out. So get on the list we'll be doing it. Um and um we

just got to set up the the money part of it and everything. I don't even know how much it's going to be. So we've got an agreement with MaxGen. Um and uh it's a validated test. So it's really we've really worked hard on it. Max has to they spent a lot of money preparing this

test. So and so um we love you guys. That's about all I can cover. I need to go get something to eat. Um I've worked all day. Thank you much. And um Jackson, thank you for doing this for us.